For decades, pancreatic cancer has defied targeted therapies by rapidly evolving escape mechanisms. In a new study, Spanish researchers report that a carefully designed three-drug strategy shut down those escape routes entirely in laboratory models, producing complete and durable tumor regression.

Traditional models of cytokinesis have long been challenged by early embryonic cell division in big, yolk-rich cells. According to recent studies, cells can divide through a dynamic interaction between cytoskeletal structures and shifting cytoplasmic material qualities rather than depending on a completely closed contractile ring.

The silent rise of antibiotic resistance could have disastrous consequences. Infections that were once curable are becoming more difficult, expensive, and occasionally uncurable.